About starling's tenet.?

hw do u explain starling's lawof tortoise heart(ventricle). hw do i discuss about it?...wats the effect of the tenet on the experiment..and also wats the effect of diastolic and systolic pressures?

starling law states that: the more increase contained by the muscle fibres, the more its power of contraction; WITHIN LIMITS (outside these boundaries the more increase the weaker contraction; ... which means over stretch & contained by this case heart failure)
it can b EXPLAINED according to molecular foundation as the muscle fibres consists of actine & myosine fibre which r present opposite respectively other. muscle contracts due to sliding of actine over myosine, due to presence of something like receptors on them; a process which uses ATP.
these two types of fibre r not perfectly matched when muscle is relaxed & shortened, but when it stretches PASSIVELY beforehand contraction, it becomes just right (each part disparate its receptor) if over stretch again they doesn't match but contained by the opposite direction)
IN UR EXPERIMENT: the stretching force of muscle (heart) is blood (venous return) power of its contraction (ejection fraction) is reflect on the blood pressure esp. systolic.
i think ur tortoise is dissected & its heart is past its sell-by date so u can apply stretching force directly through adding weights to it via a hook fixed to it (as we did while we be students)
First of all, you'll receive better responses if you type in a bearing that people who know give or take a few Starling's law will be credible to read and respond. Let's at least TRY to use English.

The easiest process to explain Starling's law (or the Frank-Starling mechanism) is this: The more you stretch a cardiac muscle fiber, the stronger it contracts. That finances, if the heart fills up beside more blood, it stretches out the muscle fibers more, and the blood will be ejected with more force than it would if the volume be lower.

I can't speak to your experiment, of course, because you didn't distribute details.

If blood is ejected at a greater force, we can expect systolic pressures to be higher when the diastolic volume (and, presumably pressure) is greater.

I hope that help.
I agree with the gas-passer on this one: if you can't agreement with the English expressions or state your experiment, it isn't likely you'll think through the Frank-Starling curve.
Up to a point, the longer the stretch of the myofibril, the greater the force of contraction. But there is a point, see in nation with CHF, where on earth you stretch so far that you "fall off" the other side of the curve, and you've gone historic the point of maximum return.
Starling`s law states " The more the initial length of the cardiac muscle fiber the more is the force of contraction".
This regulation is equally applicable to human and animal heart. Obviously within confines a dilated chamber contracts more powerfully. BP must increase.
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